Skip to main content

Management of Medical Emergency-Poisoning!

MEDICAL EMERGENCY- POISONING


  • Management of the poisoned patient involves procedures designed to prevent the absorption, minimize the toxicity, and hasten the elimination to the suspected toxin. The prompt employment of appropriate emergency management procedures often can prevent unnecessary morbidity and mortality.
  • A Regional Poison Center is a practitioner's best source of definitive treatment information and should be consulted in all poisonings, regardless of the apparent simplicity of the case.
  • In all cases, every attempt should be made to accurately identify the toxin, estimate the quantity involved, and determine the time that has passed since the exposure.
  • These data, plus patient-specific parameters such as age, weight, sex, and underlying medical condition or drug-use will assist the person and the Regional Poison Center in designing an appropriate therapeutic plan for the patient.



POISONING BY TOPICAL EXPOSURES:

  1. Immediately irrigate affected areas with a copious amount of water, use soap only if a stubborn, oily substance is the contaminant. Skin should be gently washed, not scrubbed and special attention should be given to the hairs, skin folds, umbilicus, and other areas where the contaminant might be trapped.
  2. If the patients' clothes have been contaminated, remove them during the irrigation and clean them before they are worn again or destroy them.
  3. Do not attempt to "neutralize" the contaminant with another chemical (e.g: acids and alkalis). Attempts at neutralization waste valuable time, are of no benefit, and might be harmful.
  4. Do not cover the affected area with emollients. These can trap unremoved contaminant against the skin. Severely damaged skin may be temporarily covered with a light, dry dressing.
  5. Protect yourself from contamination. Gloves, Aprons, or a change of clothes might be necessary.
  6. After the irrigation is complete, contact a Regional Poison Center for definitive treatment information.


POISONING BY EYE EXPOSURES:

  1. Immediately irrigate the eye; damage can occur within seconds. The stream of water from the tap or a pitcher should strike the patient on the forehead, temple, or bridge of the nose and then flow into the eye.
  2. The eyelids should be open, with frequent blinking during the irrigation.
  3. The irrigation should continue for at least 15 minutes (by clock) to ensure adequate removal of the contaminant and normalization of the conjunctival pH. Body temperature water or saline may be substituted for tap water as the irrigation proceeds, but only if these can be obtained without interrupting the irrigation.
  4. After the irrigation is complete, contact a Regional Poison Center for definitive-treatment information.


POISONING BY INHALATION EXPOSURES:

  1. Remove the patient from the suspected contaminated area, regardless of its apparent safety. Carbon monoxide, a common inhaled toxin, cannot be detected by sight, smell or taste.
  2. Institute artificial ventilation, if necessary, and provide supplementary humidified oxygen, if available and needed.
  3. Protect yourself from contamination at all times.
  4. Contact a Regional Poison Center for definitive treatment information.



POISONING BY INGESTIONS:

  1. Remove any remaining contaminant from inside and around the mouth of the patient.
  2. Give a small amount of water to clear the mouth and esophagus.
  3. Contact a Regional Poison Center for definitive treatment information.
  4. In many cases, it will not be necessary to take additional steps. The following information can be used if additional care is recommended by the Regional Poison Center.

Comments

Popular posts from this blog

General Mechanism of Actions of INSULIN.

GENERAL MECHANISM OF ACTION OF INSULIN AND RELATE HORMONES The body's main fuel is Glucose. Glucose is stored in the liver and muscles as Glycogen.  Excess glucose is stored in the adipose tissues which yield fatty acids via lipolysis. In the fasting state, free fatty acids supply much of the energy needs of the body except for the CNS which requires Glucose to function normally. Proteins can also be converted to glucose via gluconeogenesis. RELEASE OF INSULIN AND GLUCAGON Insulin and Glucagon are produced in the pancreas by the Islets of Langerhans . Beta cells   make up 70%-90% of the Islets and produce Insulin and Amylin while the a-cells   produce glucagon. The main function of insulin is to decrease blood glucose levels. Glucagon, along with other counter-regulatory hormones such as growth factor, cortisol, and Epinephrine, increases Blood glucose levels.  The opposing actions of Glucagon and Insulin along with other counter-regulatory hormones maintain a norma...

Diabetes Mellitus: Its Goals Of Therapy!

  GOAL OF THERPY The goals of treatment of DM includes; Reducing, controlling and managing long-term microvascular, macrovascular and neuropaathic complications. Preserving beta cell function. Preventing acute complications from increased Blood glucosel level. Minimizing hypoglycemic episodes. Maintaining patient quality of life. --Two landmark trials, THE DIABETES CONTROL AND COMPLICATION TRIAL (DCCT) and THE UNITED KINGDOM PROSPECTIVE DIABETES STUDY(UKPDS), showed that lowering blood glucose levels decreased the risk of developing chronic complications. --A near normal blood glucose level can be achieved with appropriate patient education , lifestyle modification and medications . --Proper care of DM requires goal setting and assessment for glycemic control , self-monitoring of blood glucose (SMBG) monitoring of blood glucose pressure , and lipid levels , regular monitoring for the development of complications , dietary and exercise lifestyle modifications and proper medicatio...

Diabetes Mellitus: Clinical Presentation and Diagnosis!

 CLINICAL PRESENTATION AND DIAGNOSIS OF DM 2. SCREENING American Diabetes Association (ADA) recommends routine screening for T2 DM every 3 years in all adults starting at 45 years of age. Testing of T2 DM should be considered in any adult, regardless of their age, who have a BMI greater than or equal to 25kg/m2. The ADA does not currently recommend widespread screening for T1 DM because of the relatively low incidence in the general population, although measurement of Islet antibodies may be appropriate for high-risk individuals. 3.GESTATIONAL DIABETES: "Gestational diabetes is the glucose intolerance in women during pregnancy". All pregnant women who have risk factors for T2 DM should be screened for undiagnosed T2 DM at their first prenatal visit using standard diagnostic criteria.  Any women found to have diabetes in the early point at pregnancy is considered to have T2 DM or GDM. All other pregnant women, not currently known to have DM should be screened for GDM with a 75...